[PDF][PDF] High-density lipoproteins put out the fire
Macrophages in atherosclerotic plaques are activated, inflammatory cells that directly
contribute to the disease process. De Nardo et al.(2013), now report that high-density
lipoproteins (HDL) can reprogram macrophages to be less inflammatory through an ATF3-
dependent pathway, providing another mechanistic basis for the atheroprotective properties
of HDL.
contribute to the disease process. De Nardo et al.(2013), now report that high-density
lipoproteins (HDL) can reprogram macrophages to be less inflammatory through an ATF3-
dependent pathway, providing another mechanistic basis for the atheroprotective properties
of HDL.
Macrophages in atherosclerotic plaques are activated, inflammatory cells that directly contribute to the disease process. De Nardo et al. (2013), now report that high-density lipoproteins (HDL) can reprogram macrophages to be less inflammatory through an ATF3-dependent pathway, providing another mechanistic basis for the atheroprotective properties of HDL.
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